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https://www.selleckchem.com/products/h-cys-trt-oh.html However, the extensive focus of these initiatives on data sharing does not necessarily guarantee knowledge holders' data sovereignty. To protect TEK in a context of open science more efforts should be done to operationalize traditional knowledge holders' rights to data sovereignty. To protect TEK in a context of open science more efforts should be done to operationalize traditional knowledge holders' rights to data sovereignty. Asthma is a chronic inflammatory disease, characterized by airway inflammation, hyperresponsiveness, and bronchial smooth muscle contraction. Qingfei Xiaoyan Wan (QFXYW), a traditional Chinese formula, has been shown to exert anti-asthma effects and immune response in multiple diseases. In this study, we evaluated the therapeutic mechanism of QFXYW in the suppression of allergic asthma by integrating of transcriptomics and system pharmacology. BALB/c mice were sensitized with ovalbumin (OVA) to establish the allergic asthma model, and its success was confirmed with behavioral observations. Lung histopathological analysis, inflammatory pathology scores, transcription factors were used to evaluate the effects of QFXYW on allergic asthma. The therapeutic mechanism of QFXYW in treating allergic asthma through integrated transcriptomics and system pharmacology was then determined hub genes were screened out by topological analysis and functional enrichment analysis were performed to identify key signaling paine signaling pathway. This finding will offer a novel strategy to explore effective and safe mechanism of Traditional Chinese Medicine (TCM) formula to treat allergic asthma. QFXYW may have mediated OVA-induced allergic asthma mainly through the hub genes CXCL2, CXCL1, and the cytokine signaling pathway. This finding will offer a novel strategy to explore effective and safe mechanism of Traditional Chinese Medicine (TCM) formula to treat allergic asthma. Angelica sinensis (Oliv.) Diels (AS

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