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https://www.selleckchem.com/products/2-6-dihydroxypurine.html , Fe3+ , Al3+ , Ag+ , Hg2+ , Cu2+ , Pd2+ , Zn2+ , Cr3+ , Cd2+ , Mn2+ , Ca2+ , and K+ and anions such as F- , CN- and PPi, from 2008 to 2020, because of their sensitivity and selectivity in terms of diverse colour changes for different species. This critical and comprehensive review might facilitate the improvement of more prevailing chemosensors for future exciting and broad applications. Idiopathic normal pressure hydrocephalus can present with parkinsonism. However, abnormalities of the striatal dopamine reuptake transporter are unclear. To explore presence and features of striatal dopaminergic deficit in subjects with idiopathic normal pressure hydrocephalus as compared to Parkinson's disease (PD) patients and healthy controls. We investigated 50 subjects with idiopathic normal pressure hydrocephalus, 25 with PD, and 40 healthy controls. All participants underwent [ I]-N-ω-fluoropropyl-2β-carbomethoxy-3β-(4-iodophenyl)nortropane and single-photon emission computed tomography to quantify the striatal dopamine reuptake transporter binding. All subjects with idiopathic normal pressure hydrocephalus underwent a levodopa (l-dopa) challenge test and magnetic resonance imaging to evaluate ventriculomegaly and white matter changes. Gait, cognition, balance, and continence were assessed with the Idiopathic Normal Pressure Hydrocephalus Rating Scale, and parkinsonism with the motor section ofic normal pressure hydrocephalus can present a reduction of striatal dopamine reuptake transporter binding, which is consistent with the severity of parkinsonism and qualitatively differs from that found in PD patients. Longitudinal interventional studies are needed to prove a role for striatal dopamine reuptake transporter deficit in the pathophysiology of idiopathic normal pressure hydrocephalus. © 2020 International Parkinson and Movement Disorder Society. A new sequence combining chemical-exchange saturation-transfer (CE

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