Two (25%) secondary interventions were performed for IBD-occlusion in patients with bilateral IBDs.  https://www.selleckchem.com/products/combretastatin-a4.html  The other re-intervention was a type II endoleak embolization in one of these 2 patients. The freedom from re-intervention estimate was 75% through 2 year. The overall primary assisted patency was 100% through 3 years. CONCLUSION The use of iliac branched devices in the acute setting is feasible to exclude ruptured aorto-iliac aneurysms while maintaining pelvic circulation. The secondary intervention rate is considerable, however the midterm assisted primary patency rates are promising. Further studies are needed to guide patient selection and to evaluate longer term outcomes. We present a rare case of epithelioid hemangioendothelioma arising from the wall of ulnar artery in distal forearm. The presentation was interesting in a 34-year-old man, with progressively worsening symptoms of ulnar neuropathy. A mass was seen arising from the ulnar artery on imaging with ultrasound and magnetic resonance imaging (MRI). Soft tissue epithelioid hemangioendothelioma in extremities almost always arise from the veins. Existing literature do not have elaborated imaging findings of epithelioid hemangioendothelioma arising from the arterial wall. The aim of this paper is to briefly review the interesting presentation and imaging features of this rare entity. Knowledge of such vascular tumor would avoid the mishap during surgery. Our case will add an interesting presentation of such rare pathology to the existing literature. OBJECTIVES Open conversion of endovascular aortic repair is the first-choice treatment in case of endograft failure or high-flow endoleak. However, the traditional technique based on the total removal of the endograft can produce injuries of the aortic walls, with severe consequences on the anastomoses quality. Our aim is to show the advantages of the partial endograft removal on the aortic integrity by reporting a case series including 25 delayed open conversion performed with this technique. METHODS A retrospective study was conducted over the cases of delayed open conversions performed in the last 30 months. Demographics, past medical history, endograft type, causes for conversions and early and mid-term outcomes were recorded and analyzed in relation with the technique employed (partial vs total endograft removal). RESULTS Between September 2016 and March 2019, 25 consecutive cases of EVAR failure were converted to open treatment. In all cases, the endografts were resected leaving in place part of the iliac branches, and, whenever possible, also the proximal stent of the main body. Primary technical success was achieved in 100% of cases. Disease-free survival over 18-months median follow-up was 100%. All patients underwent abdominal aortic duplex scan controls as scheduled, with no early or late post-operative complication. No anastomotic aneurysms or any surgery-related complication was observed. CONCLUSION Partial endograft removal is a safe and effective technique that could be used to protect the aortic integrity in delayed open conversions of EVAR. Budd-Chiari syndrome (BCS) is an uncommon disorder defined as an obstruction of the hepatic venous outflow. Percutaneous transluminal balloon angioplasty (PTA) is a less invasive treatment option for BCS patients. However, there are no reports regarding inferior vena cava (IVC) rupture caused by perforation route through a collateral vein during treatment of BCS. Here, we report a male patient with BCS who had a long segmental obstruction of the IVC and its collateral vessels. Here, IVC rupture occurred at the distal end of the obstructed IVC during a percutaneous angioplasty; the rupture was repaired successfully with an endovascular stent graft. BACKGROUND To investigate the correlation of clinical and ultrasound parameters with characters of vulnerable atherosclerotic carotid plaque, as evaluated at preoperative magnetic resonance angiography (MRA), in patients submitted to carotid endarterectomy (CEA), in order to develop a clinical risk score for plaque vulnerability. METHODS Preoperative data of patients submitted to CEA for significant carotid stenosis from 01/01/2012 to 31/12/2016 were retrospectively collected. The available case series was randomly divided in two groups, including a training (60%) and a validation series (40%). Data of plaque vulnerability were assessed at preoperative MRA scans. Univariate analysis was used on the training series to correlate the preoperative covariates available to the features of plaque vulnerability. Therefore, a backward selection procedure was performed again on the training series and on the validation series to assess if the same variables were associated to data of plaque vulnerability, in order to o, diabetes mellitus, coronary artery disease, neutrophil/lymphocyte ratio, platelet counts and grey-scale median value were significantly associated to the features of vulnerable plaque at preoperative MRA in patients undergoing CEA. In particular, when combined together in a "risk score", these variables provided an accurate probability of the presence of a vulnerable plaque at MRA scans. Eagle syndrome is a rare pattern of symptoms (0.16% of general population) due to the conflict with adjacent anatomical structures by an elongated styloid process or a calcified stylohyoid ligament; two variants of this condition have been described in the literature, classical and vascular. The classical form is caused by compression of the glossopharyngeal nerve and the surrounding structures from an abnormal stylohyoid apparatus, causing odynophagia and neck pain and is usually treated by otorhinolaryngologist. The vascular form, determined by the conflict between the osteo-ligamentous malformation and the extracranial carotid artery, can cause neurological symptoms due to the compression of the vessel or in some cases the dissection of the carotid artery itself. However an elongated styloid process occurs in about 4% of general population, and the most recent literature shows that the vascular form of Eagle syndrome could be an underestimated cause of carotid artery dissection (CAD) and should be considered in the differential diagnosis of this condition.